Sarkar, Sagartirtha
Sagartirtha Sarkar
VIAF ID: 3980168850990767620004 (Personal)
Permalink: http://viaf.org/viaf/3980168850990767620004
Preferred Forms
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100 0 _ ‡a Sagartirtha Sarkar
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100 1 _ ‡a Sarkar, Sagartirtha
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4xx's: Alternate Name Forms (2)
Works
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Arjunolic Acid, a Peroxisome Proliferator-Activated Receptor Alpha Agonist Regresses Cardiac Fibrosis by Inhibiting Non-canonical TGF-β Signaling |
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Comparative Proteome Profiling during Cardiac Hypertrophy and Myocardial Infarction Reveals Altered Glucose Oxidation by Differential Activation of Pyruvate Dehydrogenase E1 Component Subunit β. |
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Differential and conditional activation of PKC-isoforms dictates cardiac adaptation during physiological to pathological hypertrophy. |
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Field populations of native Indian honey bees from pesticide intensive agricultural landscape show signs of impaired olfaction. |
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Genetics fundamentals notes, 2022: |
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Hsp90/Cdc37 assembly modulates TGFβ receptor-II to act as a profibrotic regulator of TGFβ signaling during cardiac hypertrophy. |
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Improved bioavailability of targeted Curcumin delivery efficiently regressed cardiac hypertrophy by modulating apoptotic load within cardiac microenvironment |
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Influence of cytokines and growth factors in ANG II-mediated collagen upregulation by fibroblasts in rats: role of myocytes. |
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Inhibition of signal transducer and activator of transcription 3 (STAT3) attenuates interleukin-6 (IL-6)-induced collagen synthesis and resultant hypertrophy in rat heart. |
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Modulation of NFKB1/p50 by ROS leads to impaired ATP production during MI compared to cardiac hypertrophy. |
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Myocardial cell death and regeneration during progression of cardiac hypertrophy to heart failure |
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Myocyte-Derived Hsp90 Modulates Collagen Upregulation via Biphasic Activation of STAT-3 in Fibroblasts during Cardiac Hypertrophy. |
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Quantitative proteomic changes during post myocardial infarction remodeling reveals altered cardiac metabolism and Desmin aggregation in the infarct region. |
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Role of α-crystallin B as a regulatory switch in modulating cardiomyocyte apoptosis by mitochondria or endoplasmic reticulum during cardiac hypertrophy and myocardial infarction. |
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A spatio-temporal cardiomyocyte targeted vector system for efficient delivery of therapeutic payloads to regress cardiac hypertrophy abating bystander effect. |
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Upregulation of lysyl oxidase and MMPs during cardiac remodeling in human dilated cardiomyopathy |
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