Ewelina Stoczynska-Fidelus
Stoczyńska-Fidelus, Ewelina
VIAF ID: 315705109 (Personal)
Permalink: http://viaf.org/viaf/315705109
Preferred Forms
- 100 0 _ ‡a Ewelina Stoczynska-Fidelus
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4xx's: Alternate Name Forms (5)
Works
Title | Sources |
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Association between polymorphism of the DNA repair SMUG1 and UNG genes and age-related macular degeneration. | |
BCR/ABL stimulates WRN to promote survival and genomic instability. | |
Cell line with endogenous EGFRvIII expression is a suitable model for research and drug development purposes. | |
Chitin dipentanoate as the new technologically usable biomaterial. | |
Cyclic trans-phosphorylation in a homodimer as the predominant mechanism of EGFRvIII action and regulation. | |
Detection of P53 mutations in different cancer types is improved by cDNA sequencing. | |
Different mutational characteristics of TSG in cell lines and surgical specimens | |
Directed differentiation of human iPSC into insulin producing cells is improved by induced expression of PDX1 and NKX6.1 factors in IPC progenitors | |
Efficient and simple approach to in vitro culture of primary epithelial cancer cells | |
EGFR Activation Leads to Cell Death Independent of PI3K/AKT/mTOR in an AD293 Cell Line | |
EGFRvIII--a stable target for anti-EGFRvIII therapy. | |
EGFRvIII: An Oncogene with Ambiguous Role | |
The failure in the stabilization of glioblastoma-derived cell lines: spontaneous in vitro senescence as the main culprit | |
Gaps and Doubts in Search to Recognize Glioblastoma Cellular Origin and Tumor Initiating Cells | |
Generation of human iPSCs from cells of fibroblastic and epithelial origin by means of the oriP/EBNA-1 episomal reprogramming system | |
Genetic heterogeneity of RPMI-8402, a T-acute lymphoblastic leukemia cell line | |
Glioblastoma-derived spheroid cultures as an experimental model for analysis of EGFR anomalies. | |
Glioblastoma specimens with TP53 mutations do not show EGFRvIII amplification. | |
[Glioblastoma with BRAFV600E mutation and numerous metastatic foci: a case report] | |
Glioma cells showing IDH1 mutation cannot be propagated in standard cell culture conditions. | |
IDH1R132H in Neural Stem Cells: Differentiation Impaired by Increased Apoptosis. | |
Limited importance of the dominant-negative effect of TP53 missense mutations | |
Low Incidence along with Low mRNA Levels of EGFRvIII in Prostate and Colorectal Cancers Compared to Glioblastoma | |
Mechanizmy oksydacyjne w patogenezie zwyrodnienia plamki związanego z wiekiem | |
Naprawa DNA przez niehomologiczne łączenie końców : nowe białka, nowe funkcje, nowe mechanizmy | |
Phenotypical Flexibility of the EGFRvIII-Positive Glioblastoma Cell Line and the Multidirectional Influence of TGFβ and EGF on These Cells—EGFRvIII Appears as a Weak Oncogene | |
PIN3 duplication may be partially responsible for TP53 haploinsufficiency | |
Reduced expression of ELAVL4 in male meningioma patients. | |
Rola białek PAX9 i MSX1 w rozwoju i agenezji zębów | |
Screening for EGFR amplifications with a novel method and their significance for the outcome of glioblastoma patients | |
Sensitivity of neoplastic cells to senescence unveiled under standard cell culture conditions | |
SOX2 and SOX2-MYC Reprogramming Process of Fibroblasts to the Neural Stem Cells Compromised by Senescence | |
Spontaneous in vitro senescence of glioma cells confirmed by an antibody against IDH1R132H. | |
A way to understand idiopathic senescence and apoptosis in primary glioblastoma cells - possible approaches to circumvent these phenomena |