Antoine Tarjus chercheur français en physiologie et physiopathologie
Tarjus, Antoine, 1986-....
VIAF ID: 314924343 (Personal)
Permalink: http://viaf.org/viaf/314924343
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Works
Title | Sources |
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The Absence of Endothelial Sodium Channel α (αENaC) Reduces Renal Ischemia/Reperfusion Injury | |
Aldosterone promotes cardiac endothelial cell proliferation in vivo. | |
Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells | |
The Deletion of Endothelial Sodium Channel α (αENaC) Impairs Endothelium-Dependent Vasodilation and Endothelial Barrier Integrity in Endotoxemia in Vivo. | |
The diuretic torasemide does not prevent aldosterone-mediated mineralocorticoid receptor activation in cardiomyocytes | |
The endothelial αENaC contributes to vascular endothelial function in vivo. | |
The epithelial Na+ channel: a new player in the vasculature. | |
Epithelial sodium channel stiffens the vascular endothelium in vitro and in Liddle mice. | |
Genetic Deletion of Emp2 Does Not Cause Proteinuric Kidney Disease in Mice | |
Implication de la Lipocaline 2 dans les effets physiopathologiques du récepteur minéralocorticoïde dans le système cardiovasculaire. | |
Neutrophil Gelatinase-Associated Lipocalin, a Novel Mineralocorticoid Biotarget, Mediates Vascular Profibrotic Effects of Mineralocorticoids. | |
Targeting VE-PTP phosphatase protects the kidney from diabetic injury | |
Vascular mineralocorticoid receptor and blood pressure regulation |