Michael J Strong
Strong, Michael J.
VIAF ID: 31381323 (Personal)
Permalink: http://viaf.org/viaf/31381323
Preferred Forms
- 100 0 _ ‡a Michael J Strong
- 100 1 _ ‡a Strong, Michael J.
-
- 100 1 _ ‡a Strong, Michael J.
- 100 1 _ ‡a Strong, Michael J.
4xx's: Alternate Name Forms (3)
Works
Title | Sources |
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Amyotrophic lateral sclerosis and the frontotemporal dementias | |
Characterization of detergent-insoluble proteins in ALS indicates a causal link between nitrative stress and aggregation in pathogenesis. | |
Chronic aluminum-induced motor neuron degeneration: clinical, neuropathological and molecular biological aspects | |
Cognitive impairment in sporadic ALS: a pathologic continuum underlying a multisystem disorder | |
Comparative study of chronic aluminum-induced neurofilamentous aggregates with intracytoplasmic inclusions of amyotrophic lateral sclerosis. | |
Creutzfeldt-Jakob disease presenting with visual manifestations | |
Cytoplasmic sequestration of FUS/TLS associated with ALS alters histone marks through loss of nuclear protein arginine methyltransferase 1. | |
Detection of a novel frameshift mutation and regions with homozygosis within ARHGEF28 gene in familial amyotrophic lateral sclerosis | |
Diagnosis of Inclusion | |
Divergent patterns of cytosolic TDP-43 and neuronal progranulin expression following axotomy: implications for TDP-43 in the physiological response to neuronal injury | |
Dose-dependent selective suppression of light (NFL) and medium (NFM) but not heavy (NFH) molecular weight neurofilament mRNA levels in acute aluminum neurotoxicity. | |
Dysregulation of human NEFM and NEFH mRNA stability by ALS-linked miRNAs | |
The emerging role of guanine nucleotide exchange factors in ALS and other neurodegenerative diseases. | |
The evidence for altered RNA metabolism in amyotrophic lateral sclerosis (ALS). | |
Factors correlated with NPPV use in ALS | |
The frontotemporal syndromes of ALS. Clinicopathological correlates. | |
Genetic analysis of SIGMAR1 as a cause of familial ALS with dementia | |
Genetic modifiers in carriers of repeat expansions in the C9ORF72 gene | |
Hemiageusia, hemianaesthesia and hemiatrophy of the tongue | |
Human low molecular weight neurofilament (NFL) mRNA interacts with a predicted p190RhoGEF homologue (RGNEF) in humans | |
Increasing peak expiratory flow time in amyotrophic lateral sclerosis. | |
Isolation of fetal mouse motor neurons on discontinuous Percoll density gradients | |
Lack of TDP-43 abnormalities in mutant SOD1 transgenic mice shows disparity with ALS | |
Length of normal alleles of C9ORF72 GGGGCC repeat do not influence disease phenotype | |
Metabolic changes in human muscle denervation: topical 31P NMR spectroscopy studies | |
Methods for estimating numbers of motor units in biceps-brachialis muscles and losses of motor units with aging. | |
Microtubule-associated tau protein positive neuronal and glial inclusions in ALS. | |
Motor neuron disorders, c2003: | |
Multiple cerebral infarcts in patient with Moyamoya disease | |
N-butylbenzenesulphonamide, a novel neurotoxic plasticising agent | |
NIPPV: prevalence, approach and barriers to use at Canadian ALS centres. | |
Nitric oxide synthase expression in cervical spinal cord in sporadic amyotrophic lateral sclerosis. | |
NMDA induces NOS 1 translocation to the cell membrane in NGF-differentiated PC 12 cells. | |
Novel miR-b2122 regulates several ALS-related RNA-binding proteins. | |
The Ontario Neurodegenerative Disease Research Initiative (ONDRI). | |
Pathological tau deposition in Motor Neurone Disease and frontotemporal lobar degeneration associated with TDP-43 proteinopathy | |
Percutaneous endoscopic gastrostomy (PEG) in patients with ALS and bulbar dysfunction. | |
Phase II/III randomized trial of TCH346 in patients with ALS | |
Post-transcriptional control of neurofilaments in development and disease. | |
Progress in clinical neurosciences: the evidence for ALS as a multisystems disorder of limited phenotypic expression. | |
Quantitative phosphoproteomic analysis of neuronal intermediate filament proteins (NF-M/H) in Alzheimer's disease by iTRAQ | |
Recurrent encephalopathy: NAGS (N-acetylglutamate synthase) deficiency in adults. | |
Regional spread pattern predicts survival in patients with sporadic amyotrophic lateral sclerosis. | |
Reversibility of neurofilamentous inclusion formation following repeated sublethal intracisternal inoculums of AlCl3 in New Zealand white rabbits | |
RNA and Protein Interactors with TDP-43 in Human Spinal-Cord Lysates in Amyotrophic Lateral Sclerosis | |
RNA-binding proteins as molecular links between cancer and neurodegeneration. | |
RNA metabolism in ALS: when normal processes become pathological. | |
Selective loss of trans-acting instability determinants of neurofilament mRNA in amyotrophic lateral sclerosis spinal cord | |
Spinal motor neuron neuroaxonal spheroids in chronic aluminum neurotoxicity contain phosphatase-resistant high molecular weight neurofilament (NFH). | |
Strategies for Supporting Physician-Scientists in Faculty Roles: A Narrative Review With Key Informant Consultations. | |
Synergistic toxicity in an in vivo model of neurodegeneration through the co-expression of human TDP-43M337V and tauT175D protein | |
TAU mutations are not a predominant cause of frontotemporal dementia in Canadian patients | |
Tau protein aggregation in the frontal and entorhinal cortices as a function of aging. | |
Tau protein phosphorylation at Thr175 initiates fibril formation via accessibility of the N-terminal phosphatase-activating domain | |
TDP-43 aggregation inside micronuclei reveals a potential mechanism for protein inclusion formation in ALS | |
The temporal expression of amyloid precursor protein mRNA in vitro in dissociated hippocampal neuron cultures. | |
Temporal profiles of neuronal degeneration, glial proliferation, and cell death in hNFL(+/+) and NFL(-/-) mice | |
Threonine175, a novel pathological phosphorylation site on tau protein linked to multiple tauopathies | |
TMEM106B protects C9ORF72 expansion carriers against frontotemporal dementia | |
Transient middle cerebral artery occlusion induces microglial priming in the lumbar spinal cord: a novel model of neuroinflammation | |
Upregulation of GSK3beta expression in frontal and temporal cortex in ALS with cognitive impairment (ALSci) | |
Widespread cerebral haemodynamics disturbances occur early in amyotrophic lateral sclerosis. | |
Widespread neuronal and glial hyperphosphorylated tau deposition in ALS with cognitive impairment. |