Mariana J. Kaplan Mexican-American rheumatologist
Kaplan, Mariana
VIAF ID: 313286290 (Personal)
Permalink: http://viaf.org/viaf/313286290
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Works
| Title | Sources |
|---|---|
| Arthritis & Rheumatology: Evolving to Meet the Challenges of Rheumatology |
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| Association of lipoprotein subfractions and glycoprotein acetylation with coronary plaque burden in SLE |
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| Gasdermin D modulates murine lupus and its associated organ damage |
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| Heart of the matter with lupus |
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| Immunity to commensal skin fungi promotes psoriasiform skin inflammation |
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| Improved Mitochondrial Metabolism and Reduced Inflammation Following Attenuation of Murine Lupus With Coenzyme Q10 Analog Idebenone |
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| The "Infodemic" of COVID-19 |
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| Interferon lambda promotes immune dysregulation and tissue inflammation in TLR7-induced lupus |
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| Macrophage metabolic reprogramming presents a therapeutic target in lupus nephritis |
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| NETs spread ever wider in rheumatic diseases |
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| Neutrophil extracellular traps, B cells, and type I interferons contribute to immune dysregulation in hidradenitis suppurativa |
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| Neutrophil-mediated carbamylation promotes articular damage in rheumatoid arthritis |
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| Neutrophil phenotypes and functions in cancer: A consensus statement |
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| Of larks and owls |
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| Response to: 'Neutrophil extracellular traps and low-density granulocytes are associated with the interferon signature in systemic lupus erythematosus, but not in antiphospholipid syndrome' by van den Hoogen et al |
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| Sex differences in neutrophil biology modulate response to type I interferons and immunometabolism |
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| Targeting mitochondrial oxidative stress with MitoQ reduces NET formation and kidney disease in lupus-prone MRL-lpr mice |
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| Technical comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis" |
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| Transcriptomic, epigenetic, and functional analyses implicate neutrophil diversity in the pathogenesis of systemic lupus erythematosus |
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| Using the circulating proteome to assess type I interferon activity in systemic lupus erythematosus |
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| VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease |
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