Nichols, Colin G.
Colin Nichols Carl Cori Endowed Professor, and Director of the Center for Investigation of Membrane Excitability Diseases at Washington University in St. Louis, Missouri
Colin Nichols
Nichols, Colin G., 19..-....
![Sudoc [ABES], France](/viaf/images/flags/SUDOC.png "Sudoc [ABES], France")
VIAF ID: 2652516 (Personal)
Permalink: http://viaf.org/viaf/2652516
Preferred Forms
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Colin Nichols
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Colin Nichols
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Carl Cori Endowed Professor, and Director of the Center for Investigation of Membrane Excitability Diseases at Washington University in St. Louis, Missouri
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Nichols
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Colin G.
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100 1 _ ‡a Nichols, Colin G.
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100 1 _ ‡a Nichols, Colin G.
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Nichols, Colin G.
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Nichols, Colin G.,
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19..-....
4xx's: Alternate Name Forms (2)
Works
| Title | Sources |
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| ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9 |
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| Atomistic basis of opening and conduction in mammalian inward rectifier potassium (Kir2.2) channels |
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| Beta-cell excitability and excitability-driven diabetes in adult Zebrafish islets |
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| Control of Kir channel gating by cytoplasmic domain interface interactions |
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| Expression and function of ATP-dependent potassium channels in zebrafish islet β-cells. |
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| Glibenclamide reverses cardiovascular abnormalities of Cantu syndrome driven by KATP channel overactivity |
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| Glibenclamide treatment in a Cantú syndrome patient with a pathogenic ABCC9 gain-of-function variant: Initial experience |
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| In vivo monitoring of intracellular Ca dynamics in the pancreatic β-cells of zebrafish embryos |
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| Ion channel localization methods and protocols |
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| Kir6.1-dependent KATP channels in lymphatic smooth muscle and vessel dysfunction in mice with Kir6.1 gain-of-function |
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| Macrophage secretion of miR-106b-5p causes renin-dependent hypertension |
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| Potassium channel selectivity filter dynamics revealed by single-molecule FRET |
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| Preferential Gq signaling in diabetes: an electrical switch in incretin action and in diabetes progression? |
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| The role of membrane excitability in pancreatic β-cell glucotoxicity |
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