Pierre-Olivier Frappart
Frappart, Pierre-Olivier
VIAF ID: 2590159109698806900003 (Personal)
Permalink: http://viaf.org/viaf/2590159109698806900003
Preferred Forms
- 100 1 _ ‡a Frappart, Pierre-Olivier
- 100 0 _ ‡a Pierre-Olivier Frappart
4xx's: Alternate Name Forms (2)
5xx's: Related Names (3)
- 510 2 _ ‡a Deutsches Krebsforschungszentrum ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 510 2 _ ‡a Universität Ulm ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 510 2 _ ‡a Universitätsmedizin Mainz ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
Works
Title | Sources |
---|---|
Ataxia-telangiectasia and related diseases | |
ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage. | |
ATR maintains select progenitors during nervous system development | |
ATRIP protects progenitor cells against DNA damage in vivo | |
BRCA2 function and the central nervous system. | |
BRCA2 is required for neurogenesis and suppression of medulloblastoma | |
Conditional deletion of Nbs1 in murine cells reveals its role in branching repair pathways of DNA double-strand breaks | |
Epidermal Nbn deletion causes premature hair loss and a phenotype resembling psoriasiform dermatitis | |
An essential function for NBS1 in the prevention of ataxia and cerebellar defects | |
Glucokinase is highly induced and glucose-6-phosphatase poorly repressed in liver of rainbow trout (Oncorhynchus mykiss) by a single meal with glucose | |
Identification of differential gene expression in in vitro FSH treated pig granulosa cells using suppression subtractive hybridization. | |
An inducible null mutant murine model of Nijmegen breakage syndrome proves the essential function of NBS1 in chromosomal stability and cell viability. | |
MCPH1 regulates the neuroprogenitor division mode by coupling the centrosomal cycle with mitotic entry through the Chk1-Cdc25 pathway | |
Mouse models of DNA double-strand break repair and neurological disease | |
Nbn and atm cooperate in a tissue and developmental stage-specific manner to prevent double strand breaks and apoptosis in developing brain and eye. | |
Nbn heterozygosity renders mice susceptible to tumor formation and ionizing radiation-induced tumorigenesis | |
Neurofibromin specific antibody differentiates malignant peripheral nerve sheath tumors (MPNST) from other spindle cell neoplasms | |
Nibrin functions in Ig class-switch recombination | |
A novel function of DNA repair molecule Nbs1 in terminal differentiation of the lens fibre cells and cataractogenesis. | |
Pancreatic Ductal Adenocarcinoma (PDAC) Organoids: The Shining Light at the End of the Tunnel for Drug Response Prediction and Personalized Medicine | |
Pancreatic Ductal Organoids React Kras Dependent to the Removal of Tumor Suppressive Roadblocks | |
Poly(ADP-ribose) polymerase-1, a novel partner of progesterone receptors in endometrial cancer and its precursors | |
Precision medicine meets the DNA damage response in pancreatic cancer. | |
Progenitor death drives retinal dysplasia and neuronal degeneration in a mouse model of Atrip-Seckel syndrome | |
Recurrent genomic alterations characterize medulloblastoma arising from DNA double-strand break repair deficiency | |
RINT1 functions as a multitasking protein at the crossroads between genomic stability, ER homeostasis, and autophagy | |
Rint1 inactivation triggers genomic instability, ER stress and autophagy inhibition in the brain. | |
Synergistic targeting and resistance to PARP inhibition in DNA damage repair-deficient pancreatic cancer |