Bernhardi, Rommy von
Rommy von Bernhardi researcher ORCID ID = 0000-0002-9022-7676
VIAF ID: 16686067 (Personal)
Permalink: http://viaf.org/viaf/16686067
Preferred Forms
-
- 100 1 _ ‡a Bernhardi, Rommy von
-
- 100 1 _ ‡a Bernhardi, Rommy von
- 100 1 _ ‡a Bernhardi, Rommy von
-
- 100 1 _ ‡a Bernhardi, Rommy von
- 100 0 _ ‡a Rommy von Bernhardi ‡c researcher ORCID ID = 0000-0002-9022-7676
4xx's: Alternate Name Forms (4)
5xx's: Related Names (2)
Works
Title | Sources |
---|---|
Acetylcholinesterase induces the expression of the beta-amyloid precursor protein in glia and activates glial cells in culture. | |
Age-dependent changes on TGFβ1 Smad3 pathway modify the pattern of microglial cell activation | |
Alzheimer's disease: redox dysregulation as a common denominator for diverse pathogenic mechanisms. | |
Astrocytes inhibit nitric oxide-dependent Ca(2+) dynamics in activated microglia: involvement of ATP released via pannexin 1 channels. | |
ATP is required and advances cytokine-induced gap junction formation in microglia in vitro. | |
De-automatization through awareness of subjective realism : a neurophenomenological account of meditative states and their effect on cognitive bias | |
Aβ potentiates inflammatory activation of glial cells induced by scavenger receptor ligands and inflammatory mediators in culture. | |
[Biological mechanisms involved in the spread of traumatic brain damage] | |
c-Abl tyrosine kinase modulates tau pathology and Cdk5 phosphorylation in AD transgenic mice | |
Commentaries on Viewpoint: The ongoing need for good physiological investigation: Obstructive sleep apnea in HIV patients as a paradigm | |
Connexin43 hemichannels mediate secondary cellular damage spread from the trauma zone to distal zones in astrocyte monolayers. | |
D-serine released by astrocytes in brainstem regulates breathing response to CO2 levels | |
Désautomatisation à travers la prise de conscience du réalisme subjectif : exploration neurophénoménologique des états méditatifs et de leurs effets sur le biais cognitif. | |
Glial Cells and Integrity of the Nervous System. | |
Glial Cells in Health and Disease of the CNS | |
Immobilized amyloid precursor protein constructs: a tool for the in vitro screening of glial cell reactivity | |
Immunotherapy in Alzheimer's disease: where do we stand? Where should we go? | |
Impact of Aging in Microglia-Mediated D-Serine Balance in the CNS | |
Is the supply of axoplasmic proteins a burden for the cell body? Morphometry of sensory neurons and amino acid incorporation into their cell bodies. | |
Microglia Function in the Normal Brain. | |
Microglial cell dysregulation in brain aging and neurodegeneration | |
Microglial reactivity to beta-amyloid is modulated by astrocytes and proinflammatory factors | |
Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease. | |
Neurodegenerative diseases, c2007: | |
Oligodendrocytes damage in Alzheimer's disease: beta amyloid toxicity and inflammation. | |
Phosphorylated tau potentiates Aβ-induced mitochondrial damage in mature neurons. | |
The Plastic Brain | |
Prenatal to early postnatal nicotine exposure impairs central chemoreception and modifies breathing pattern in mouse neonates: a probable link to sudden infant death syndrome. | |
Protection of rat primary hippocampal cultures from A beta cytotoxicity by pro-inflammatory molecules is mediated by astrocytes. | |
Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition. | |
Role of scavenger receptors in glia-mediated neuroinflammatory response associated with Alzheimer's disease. | |
Role of TGFβ signaling in the pathogenesis of Alzheimer's disease. | |
Scavenger Receptor-A deficiency impairs immune response of microglia and astrocytes potentiating Alzheimer's disease pathophysiology. | |
SR-A regulates the inflammatory activation of astrocytes | |
Thiazolidinediones promote axonal growth through the activation of the JNK pathway | |
Transforming growth factor-beta 1 produced by hippocampal cells modulates microglial reactivity in culture. | |
Transforming growth factor β1 modulates amyloid β-induced glial activation through the Smad3-dependent induction of MAPK phosphatase-1 | |
Understanding risk factors for Alzheimer's disease: interplay of neuroinflammation, connexin-based communication and oxidative stress. | |
What Is Neural Plasticity? |