Reaman, Gregory H.
![Sudoc [ABES], France](/viaf/images/flags/SUDOC.png "Sudoc [ABES], France")
Reaman, Gregory H. 19..-...
Gregory H. Reaman
VIAF ID: 161041385 (Personal)
Permalink: http://viaf.org/viaf/161041385
Preferred Forms
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Gregory H. Reaman
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Reaman, Gregory H.
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100 1 _ ‡a Reaman, Gregory H.
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100 1 _
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Reaman, Gregory H.
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100 1 _
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Reaman, Gregory H.
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100 1 _ ‡a Reaman, Gregory H.
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100 1 _
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Reaman, Gregory H.
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100 1 _ ‡a Reaman, Gregory H.
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100 1 _ ‡a Reaman, Gregory H. ‡d (19..- ).
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100 1 _
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Reaman, Gregory H.
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19..-...
4xx's: Alternate Name Forms (3)
5xx's: Related Names (5)
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Children's National Medical Center
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Children's National Medical Center
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Washington, DC
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Affiliation
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George Washington University
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George Washington University
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Affiliation
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Washington, DC
Works
| Title | Sources |
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| Bone Marrow Failure |
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| Brain Tumors in Children |
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| Childhood Leukemia A Practical Handbook |
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| The Critical Role of Academic Clinical Trials in Pediatric Cancer Drug Approvals: Design, Conduct, and Fit for Purpose Data for Positive Regulatory Decisions |
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| Genome-wide association study identifies germline polymorphisms associated with relapse of childhood acute lymphoblastic leukemia |
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| Hematopoietic Cell Transplantation in Children with Cancer |
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| Identification of novel cluster groups in pediatric high-risk B-precursor acute lymphoblastic leukemia with gene expression profiling: correlation with genome-wide DNA copy number alterations, clinical characteristics, and outcome |
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| Intensified chemotherapy without SCT in infant ALL: results from COG P9407 (Cohort 3) |
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| Key pathways are frequently mutated in high-risk childhood acute lymphoblastic leukemia: a report from the Children's Oncology Group |
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| Outcome modeling with CRLF2, IKZF1, JAK, and minimal residual disease in pediatric acute lymphoblastic leukemia: a Children's Oncology Group study |
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| Pediatric Cancer Therapeutics Development |
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| Potent obatoclax cytotoxicity and activation of triple death mode killing across infant acute lymphoblastic leukemia |
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| Rearrangement of CRLF2 is associated with mutation of JAK kinases, alteration of IKZF1, Hispanic/Latino ethnicity, and a poor outcome in pediatric B-progenitor acute lymphoblastic leukemia |
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