Jean Langhorne
Langhorne, Jean
Langhorne, J. (Jean)
Langhorne, J.
VIAF ID: 15056961 (Personal)
Permalink: http://viaf.org/viaf/15056961
Preferred Forms
- 100 0 _ ‡a Jean Langhorne
- 200 _ | ‡a Langhorne ‡b Jean
-
- 100 1 _ ‡a Langhorne, J. ‡q (Jean)
-
- 100 1 _ ‡a Langhorne, J. ‡q (Jean)
-
- 100 1 _ ‡a Langhorne, Jean
-
- 100 1 _ ‡a Langhorne, Jean
- 100 1 _ ‡a Langhorne, Jean
4xx's: Alternate Name Forms (4)
5xx's: Related Names (3)
- 551 _ _ ‡a London ‡4 ortw ‡4 https://d-nb.info/standards/elementset/gnd#placeOfActivity
- 510 2 _ ‡a National Institute for Medical Research
- 510 2 _ ‡a National Institute for Medical Research ‡g London ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
Works
Title | Sources |
---|---|
IFNγ and IL-12 Restrict Th2 Responses during Helminth/Plasmodium Co-Infection and Promote IFNγ from Th2 Cells | |
The immune response to Plasmodium chabaudi malaria in interleukin-4-deficient mice. | |
Immune status of a mu, kappa transgenic mouse line. Deficient response to bacterially related antigens | |
Immunity against HIV/AIDS, Malaria, and Tuberculosis during Co-Infections with Neglected Infectious Diseases: Recommendations for the European Union Research Priorities | |
Immunology and immunopathogenesis of malaria | |
Immunopathologie des infections Plasmodium chabaudi virulentes et non virulentes dans les souris C57BL/6 | |
In vivo approaches reveal a key role for DCs in CD4+ T cell activation and parasite clearance during the acute phase of experimental blood-stage malaria | |
Individual-level variations in malaria susceptibility and acquisition of clinical protection | |
Induction of an IL7-R(+)c-Kit(hi) myelolymphoid progenitor critically dependent on IFN-gamma signaling during acute malaria. | |
Induction of T cell activity in athymic (nu/nu) mice infected with Trypanosoma rhodesiense | |
Influence of Plasmodium chabaudi adami on the isotypic distribution of the antibody response of mice to sheep erythrocytes | |
Limiting dilution analysis of antigen-specific CD4+ T-cell responses in mice. | |
Low CD4 | |
Low CD4(+) T cell responses to the C-terminal region of the malaria merozoite surface protein-1 may be attributed to processing within distinct MHC class II pathways. | |
Malaria infection changes the ability of splenic dendritic cell populations to stimulate antigen-specific T cells | |
Malaria infection induces virus expression in human immunodeficiency virus transgenic mice by CD4 T cell-dependent immune activation | |
Malaria-specific transgenic CD4(+) T cells protect immunodeficient mice from lethal infection and demonstrate requirement for a protective threshold of antibody production for parasite clearance. | |
Malarial anemia: of mice and men. | |
Medial histocompatibility antigens. | |
Memory B-cell and antibody responses induced by Plasmodium falciparum sporozoite immunization | |
Molecular approaches to malaria. | |
The molecular interactions with helper T cells which limit antigen-specific B cell differentiation | |
Mosquitoes Reset Malaria Parasites | |
Pathology of Plasmodium chabaudi chabaudi infection and mortality in interleukin-10-deficient mice are ameliorated by anti-tumor necrosis factor alpha and exacerbated by anti-transforming growth factor beta antibodies | |
The pathology of Plasmodium chabaudi infection is not ameliorated by the secreted filarial nematode immunomodulatory molecule, ES-62. | |
Phenotypic and functional profiling of CD4 T cell compartment in distinct populations of healthy adults with different antigenic exposure | |
The pir multigene family of Plasmodium: antigenic variation and beyond | |
The plasma concentration of the B cell activating factor is increased in children with acute malaria | |
Plasmodium chabaudi chabaudi: differential susceptibility of gene-targeted mice deficient in IL-10 to an erythrocytic-stage infection. | |
Plasmodium chabaudi chabaudi infection in mice induces strong B cell responses and striking but temporary changes in splenic cell distribution | |
Plasmodium falciparum STEVOR proteins are highly expressed in patient isolates and located in the surface membranes of infected red blood cells and the apical tips of merozoites | |
Plasmodium knowlesi in the marmoset (Callithrix jacchus). | |
Priming of CD4+ T cells and development of CD4+ T cell memory; lessons for malaria | |
Production of murine interleukin-4 and interleukin-5 by recombinant baculovirus. | |
PSLAP, a protein with multiple adhesive motifs, is expressed in Plasmodium falciparum gametocytes | |
Quantitative analysis of the response of human T cell receptor Vγ9+ cells toPlasmodium falciparum | |
Quantitative studies on concanavalin A-induced, TCGF-reactive T cells. I. Correlation between proliferation and lectin-dependent cytolytic activity | |
Rapid changes in transcription profiles of the Plasmodium yoelii yir multigene family in clonal populations: lack of epigenetic memory? | |
Recent advances in malaria research: new tools to understand an old enemy | |
Recovery of an antiviral antibody response following attrition caused by unrelated infection | |
Regulation of immune response by Plasmodium-infected red blood cells. | |
The relevance of non-human primate and rodent malaria models for humans | |
Replication of Plasmodium in reticulocytes can occur without hemozoin formation, resulting in chloroquine resistance | |
The response of gamma delta T cells in malaria infections: a hypothesis. | |
Restoration of pattern recognition receptor costimulation to treat chromoblastomycosis, a chronic fungal infection of the skin | |
The role of animal models for research on severe malaria | |
Role of gamma interferon during infection with Plasmodium chabaudi chabaudi. | |
Role of non-H-2 antigens in the cytotoxic T cell response to allogeneic H-2. | |
The role of the spleen in malaria | |
The roles of cytokines produced in the immune response to the erythrocytic stages of mouse malarias | |
Screening cDNA expression libraries in lambda gt11 with a T cell hybridoma | |
Sheep gastric mucins as a source of blood-group-I and -i antigens | |
Signatures of malaria-associated pathology revealed by high-resolution whole-blood transcriptomics in a rodent model of malaria | |
Studies on the heterophile antibodies of infectious mononucleosis. I. Separation of four antibody populations, one of which contains lymphocytotoxic activity | |
Systemic lupus erythematosus-associated defects in the inhibitory receptor FcgammaRIIb reduce susceptibility to malaria. | |
T cell-derived IL-10 and its impact on the regulation of host responses during malaria | |
T Cell-Derived IL-10 Impairs Host Resistance to Mycobacterium tuberculosis Infection | |
T-cell immunity to malaria | |
Targeted delivery of antigen processing inhibitors to antigen presenting cells via mannose receptors. | |
Targeting cytotoxic T cells to antigen-specific B lymphocytes | |
Transcriptomic profiling of microglia reveals signatures of cell activation and immune response, during experimental cerebral malaria | |
Transcriptomics identified a critical role for Th2 cell-intrinsic miR-155 in mediating allergy and antihelminth immunity | |
Transformation of the rodent malaria parasite Plasmodium chabaudi | |
Transient deficiency of dendritic cells results in lack of a merozoite surface protein 1-specific CD4 T cell response during peak Plasmodium chabaudi blood-stage infection | |
Trimethoprim-Sulfamethoxazole Prophylaxis During Live Malaria Sporozoite Immunization Induces Long-Lived, Homologous, and Heterologous Protective Immunity Against Sporozoite Challenge | |
Tumor necrosis factor alpha p55 receptor is important for development of memory responses to blood-stage malaria infection | |
Vector transmission regulates immune control of Plasmodium virulence |