Vilhelm A. Bohr forsker, National Institute on Aging
Bohr, Vilhelm A.
![Sudoc [ABES], France](/viaf/images/flags/SUDOC.png "Sudoc [ABES], France")
Bohr, V.A. (Vilhelm A.)
VIAF ID: 13991635 ( Personal )
Permalink: http://viaf.org/viaf/13991635
Preferred Forms
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100 1 _ ‡a Bohr, V.A. ‡q (Vilhelm A.)
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100 1 _ ‡a Bohr, Vilhelm A
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100 1 _
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Bohr, Vilhelm A
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100 1 _
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Bohr, Vilhelm A.
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100 1 0
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Bohr, Vilhelm A.
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100 1 _ ‡a Bohr, Vilhelm A.
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100 1 _
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Bohr, Vilhelm A.
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100 1 _ ‡a Bohr, Vilhelm A.
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100 1 _
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Bohr, Vilhelm A.
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100 0 _
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Vilhelm A. Bohr
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forsker, National Institute on Aging
4xx's: Alternate Name Forms (4)
Works
| Title | Sources |
|---|---|
| Bohr på ny |
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| DNA repair mechanisms |
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| Heterochromatin: an epigenetic point of view in aging |
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| Livets kode: DNA |
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| Mitochondrial and nuclear DNA-repair capacity of various brain regions in mouse is altered in an age-dependent manner. |
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| Mitochondrial repair of 8-oxoguanine is deficient in Cockayne syndrome group B. |
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| Mitophagy and Alzheimer's Disease: Cellular and Molecular Mechanisms. |
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| Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer's disease |
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| Modulation of Werner syndrome protein function by a single mutation in the conserved RecQ domain |
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| Molecular biology of aging : proceedings of a symposium held at the Royal Danish academy of sciences and letters, June 14-18, 1998 |
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| Multiple interaction partners for Cockayne syndrome proteins: implications for genome and transcriptome maintenance |
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| Multiple RPAs make WRN syndrome protein a superhelicase. |
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| NAD+ augmentation restores mitophagy and limits accelerated aging in Werner syndrome |
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| NAD+ in Aging: Molecular Mechanisms and Translational Implications. |
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| NAD+ Replenishment Improves Lifespan and Healthspan in Ataxia Telangiectasia Models via Mitophagy and DNA Repair. |
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| NAD+ supplementation normalizes key Alzheimer's features and DNA damage responses in a new AD mouse model with introduced DNA repair deficiency. |
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| NAD+: The convergence of DNA repair and mitophagy. |
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| Negative regulation of STAT3 protein-mediated cellular respiration by SIRT1 protein |
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| NEIL1 stimulates neurogenesis and suppresses neuroinflammation after stress |
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| Neurons efficiently repair glutamate-induced oxidative DNA damage by a process involving CREB-mediated up-regulation of apurinic endonuclease 1. |
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| New areas of focus at workshop on human diseases involving DNA repair deficiency and premature aging. |
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| Nicotinamide Improves Aspects of Healthspan, but Not Lifespan, in Mice. |
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| Novel DNA mismatch-repair activity involving YB-1 in human mitochondria |
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| Nuclear DNA damage signalling to mitochondria in ageing |
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| Nucleolin inhibits G4 oligonucleotide unwinding by Werner helicase |
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| Nutrient-sensitive mitochondrial NAD+ levels dictate cell survival |
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| Nutrition and aging |
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| Oxidative damage in telomeric DNA disrupts recognition by TRF1 and TRF2 |
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| Oxidative stress and mitochondrial DNA repair: implications for NRTIs induced DNA damage |
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| Oxidized guanine lesions and hOgg1 activity in lung cancer. |
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| P VI.4 Studies on the possible detection of drug-induced topoiso-merase I-DNA complexes by nucleotide excision repair |
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| p53 functions in the incorporation step in DNA base excision repair in mouse liver mitochondria. |
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| Partial loss of the DNA repair scaffolding protein, Xrcc1, results in increased brain damage and reduced recovery from ischemic stroke in mice |
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| Pathways defective in the human premature aging disease Werner syndrome |
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| Phenotypic consequences of mutations in the conserved motifs of the putative helicase domain of the human Cockayne syndrome group B gene. |
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| Phosphorylation of human oxoguanine DNA glycosylase (alpha-OGG1) modulates its function |
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| POT1 stimulates RecQ helicases WRN and BLM to unwind telomeric DNA substrates |
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| A potential impact of DNA repair on ageing and lifespan in the ageing model organism Podospora anserina: decrease in mitochondrial DNA repair activity during ageing |
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| Preferential DNA repair in active genes |
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| Primary fibroblasts of Cockayne syndrome patients are defective in cellular repair of 8-hydroxyguanine and 8-hydroxyadenine resulting from oxidative stress |
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| Protecting the mitochondrial powerhouse. |
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| Quantitative analysis of WRN exonuclease activity by isotope dilution mass spectrometry |
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| RAPADILINO RECQL4 mutant protein lacks helicase and ATPase activity |
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| Re-equilibration of imbalanced NAD metabolism ameliorates the impact of telomere dysfunction |
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| Recent Advances in Understanding Werner Syndrome. |
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| RECQ helicase RECQL4 participates in non-homologous end joining and interacts with the Ku complex |
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| RECQ1 is required for cellular resistance to replication stress and catalyzes strand exchange on stalled replication fork structures |
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| RECQL4 localizes to mitochondria and preserves mitochondrial DNA integrity |
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| RECQL4, the protein mutated in Rothmund-Thomson syndrome, functions in telomere maintenance |
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| RECQL5 cooperates with Topoisomerase II alpha in DNA decatenation and cell cycle progression |
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| RECQL5 has unique strand annealing properties relative to the other human RecQ helicase proteins |
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| RECQL5 plays co-operative and complementary roles with WRN syndrome helicase |
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| Recruitment and retention dynamics of RECQL5 at DNA double strand break sites. |
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| Regulation of the human Suv3 helicase on DNA by inorganic cofactors |
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| Regulation of WRN helicase activity in human base excision repair. |
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| Regulatory interplay of Cockayne syndrome B ATPase and stress-response gene ATF3 following genotoxic stress |
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| Relationships between human vitality and mitochondrial respiratory parameters, reactive oxygen species production and dNTP levels in peripheral blood mononuclear cells. |
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| Repair of formamidopyrimidines in DNA involves different glycosylases: role of the OGG1, NTH1, and NEIL1 enzymes. |
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| Repair of persistent strand breaks in the mitochondrial genome |
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| A research agenda for aging in China in the 21st century. |
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| Rev1 contributes to proper mitochondrial function via the PARP-NAD+-SIRT1-PGC1α axis |
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| The role of DNA damage and repair in cell aging |
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| Roles of RECQ helicases in recombination based DNA repair, genomic stability and aging |
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| Roles of the Werner syndrome protein in pathways required for maintenance of genome stability |
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| Sarcopenia, aging and prospective interventional strategies. |
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| Senolytic therapy alleviates Aβ-associated oligodendrocyte progenitor cell senescence and cognitive deficits in an Alzheimer's disease model |
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| Short-term NAD+ supplementation prevents hearing loss in mouse models of Cockayne syndrome |
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| Single-molecule imaging reveals a common mechanism shared by G-quadruplex-resolving helicases |
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| Site-specific noncovalent interaction of the biopolymer poly(ADP-ribose) with the Werner syndrome protein regulates protein functions. |
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| Skin Abnormalities in Disorders with DNA Repair Defects, Premature Aging, and Mitochondrial Dysfunction |
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| Slow mitochondrial repair of 5'-AMP renders mtDNA susceptible to damage in APTX deficient cells. |
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| SLX4 contributes to telomere preservation and regulated processing of telomeric joint molecule intermediates |
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| Spatial Transcriptomics Reveals Genes Associated with Dysregulated Mitochondrial Functions and Stress Signaling in Alzheimer Disease |
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| Special issue on the segmental progeria Cockayne syndrome |
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| Sporadic Alzheimer disease fibroblasts display an oxidative stress phenotype |
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| SRT1720 improves survival and healthspan of obese mice |
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| Stimulation of flap endonuclease-1 by the Bloom's syndrome protein |
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| Substrate specific stimulation of NEIL1 by WRN but not the other human RecQ helicases |
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| Telomere repeat binding factor 2 interacts with base excision repair proteins and stimulates DNA synthesis by DNA polymerase beta |
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| Telomere, telomerase and aging |
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| Telomeric D-loops containing 8-oxo-2'-deoxyguanosine are preferred substrates for Werner and Bloom syndrome helicases and are bound by POT1 |
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