Peter F Zipfel researcher
Zipfel, Peter F. 1954-
Zipfel, Peter F.
Zipfel, P.F.
VIAF ID: 12635440 (Personal)
Permalink: http://viaf.org/viaf/12635440
Preferred Forms
- 100 0 _ ‡a Peter F Zipfel ‡c researcher
- 200 _ | ‡a Zipfel ‡b Peter F.
- 100 1 _ ‡a Zipfel, P.F.
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- 100 1 _ ‡a Zipfel, Peter F.
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- 100 1 _ ‡a Zipfel, Peter F.
- 100 1 _ ‡a Zipfel, Peter F. ‡d 1954-
4xx's: Alternate Name Forms (4)
5xx's: Related Names (8)
- 510 2 _ ‡a Bremen, Univ
- 510 2 _ ‡a Friedrich-Schiller-Universität Jena ‡b Institut für Mikrobiologie ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 510 2 _ ‡a Friedrich-Schiller-Universität Jena ‡b Professur für Infektionsbiologie ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 510 2 _ ‡a Hans-Knöll-Institut für Naturstoff-Forschung ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 510 2 _ ‡a Hans-Knöll-Institut für Naturstoff-Forschung
- 551 _ _ ‡a Jena ‡4 ortw ‡4 https://d-nb.info/standards/elementset/gnd#placeOfActivity
- 510 2 _ ‡a Leibniz-Institut für Naturstoff-Forschung und Infektionsbiologie ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
- 500 1 _ ‡a Skerka, Christine ‡4 bezf ‡4 https://d-nb.info/standards/elementset/gnd#familialRelationship ‡e Beziehung familiaer
Works
Title | Sources |
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Binding of the SARS-CoV-2 Spike Protein to Platelet Factor 4 A Proposed Mechanism for the Generation of Pathogenic Antibodies | |
Borrelia recurrentis employs a novel multifunctional surface protein with anti-complement, anti-opsonic and invasive potential to escape innate immunity | |
Borrelia valaisiana resist complement-mediated killing independently of the recruitment of immune regulators and inactivation of complement components | |
Candida albicans Tef1 bindet an Dektin-1 und Dektin-2 auf humanen Dendritischen Zellen und löst eine inflammatorische Immunantwort aus | |
Complement and kidney disease | |
Conserved patterns of microbial immune escape: pathogenic microbes of diverse origin target the human terminal complement inhibitor vitronectin via a single common motif | |
Contribution of the infection-associated complement regulator-acquiring surface protein 4 (ErpC) to complement resistance of Borrelia burgdorferi | |
Elucidating the immune evasion mechanisms of borrelia mayonii, the causative agent of lyme disease | |
FHR-4A: a new factor H-related protein is encoded by the human FHR-4 gene. | |
FHR5 Binds to Laminins, Uses Separate C3b and Surface-Binding Sites, and Activates Complement on Malondialdehyde-Acetaldehyde Surfaces. | |
Functional characterization of Borrelia spielmanii outer surface proteins that interact with distinct members of the human factor H protein family and with plasminogen | |
Funktion von Ssl11 und PurA von Staphylococcus aureus bei der Komplementevasion | |
Generation and functional in vivo characterization of a lipid kinase defective phosphatidylinositol 3-kinase Vps34p of Candida albicans. | |
Genetic Factors of the Disease Course After Sepsis: Rare Deleterious Variants Are Predictive | |
Genetic influences on plasma CFH and CFHR1 concentrations and their role in susceptibility to age-related macular degeneration | |
Haemophilus influenzae protein E recognizes the C-terminal domain of vitronectin and modulates the membrane attack complex | |
Haemophilus influenzae uses the surface protein E to acquire human plasminogen and to evade innate immunity. | |
HED | |
The host immune regulator factor H interacts via two contact sites with the PspC protein of Streptococcus pneumoniae and mediates adhesion to host epithelial cells | |
Host-pneumococcal interactions - from the lung to the brain | |
human complement system a mathematical analysis of its regulation | |
Hyperspectral imaging using intracellular spies : quantitative real-time measurement of intracelluar parameters in vivo during host-pathogen interaction | |
An imbalance of human complement regulatory proteins CFHR1, CFHR3 and factor H influences risk for age-related macular degeneration (AMD) | |
Immune evasion of Moraxella catarrhalis involves ubiquitous surface protein A-dependent C3d binding. | |
Immune evasion of the human pathogenic yeast Candida albicans: Pra1 is a Factor H, FHL-1 and plasminogen binding surface protein | |
Immune modulation by complement receptor 3-dependent human monocyte TGF-β1-transporting vesicles | |
In situ Bildgebung der AA-Amyloidose mithilfe eines auf B10 basierenden VHH-Antikörpers | |
Inadequate binding of immune regulator factor H is associated with sensitivity of Borrelia lusitaniae to human complement | |
Interaction of Shiga toxin 2 with complement regulators of the factor H protein family | |
An interactive web database of factor H-associated hemolytic uremic syndrome mutations: insights into the structural consequences of disease-associated mutations | |
intraphagocytic long-term survival of the mucormycotic agent Lichtheimia corymbifera | |
Kallikrein Cleaves C3 and Activates Complement. | |
Leptospiral endostatin-like protein A is a bacterial cell surface receptor for human plasminogen | |
LfhA, a novel factor H-binding protein of Leptospira interrogans | |
Mechanismen der Kontaktaktivierung während einer Infektion mit Streptococcus pyogenes | |
Membranoproliferative glomerulonephritis and C3 glomerulopathy in children: change in treatment modality? : a report of a case series | |
The molecular basis for hereditary porcine membranoproliferative glomerulonephritis type II: point mutations in the factor H coding sequence block protein secretion | |
Monomeric C-reactive protein modulates classic complement activation on necrotic cells | |
Moraxella catarrhalis Binds Plasminogen To Evade Host Innate Immunity | |
Mutations in FN1 cause glomerulopathy with fibronectin deposits. | |
The mycota : a comprehensive treatise on fungi as experimental systems for basic and applied research | |
New approaches to the treatment of dense deposit disease | |
New insights into disease-specific absence of complement factor H related protein C in mouse models of spontaneous autoimmune diseases | |
A novel antibody against human properdin inhibits the alternative complement system and specifically detects properdin from blood samples. | |
Onchocerca volvulus microfilariae avoid complement attack by direct binding of factor H | |
The opportunistic human pathogenic fungus Aspergillus fumigatus evades the host complement system | |
The pH-regulated antigen 1 of Candida albicans binds the human complement inhibitor C4b-binding protein and mediates fungal complement evasion | |
Pig complement regulator factor H: molecular cloning and functional characterization. | |
Protein load impairs factor H binding promoting complement-dependent dysfunction of proximal tubular cells | |
Pseudomonas aeruginosa Uses Dihydrolipoamide Dehydrogenase (Lpd) to Bind to the Human Terminal Pathway Regulators Vitronectin and Clusterin to Inhibit Terminal Pathway Complement Attack | |
Publisher Correction: ApoE attenuates unresolvable inflammation by complex formation with activated C1q | |
The putative vacuolar ATPase subunit Vma7p of Candida albicans is involved in vacuole acidification, hyphal development and virulence | |
Recombinant production of MFHR1, a novel synthetic multitarget complement inhibitor, in moss bioreactors | |
The role of defective complement control in hemolytic uremic syndrome | |
Role of the Vps34p-interacting protein Ade5,7p in hyphal growth and virulence of Candida albicans | |
Rolle des Komplementaktivators FHR5 in der Immunhomöostase und bei der Pathogenese der Nierenerkrankung C3-Glomerulopathie : wenn der Freund zum Gegner wird | |
Secreted Aspergillus fumigatus protease Alp1 degrades human complement proteins C3, C4, and C5. | |
The secreted Candida albicans protein Pra1 disrupts host defense by broadly targeting and blocking complement C3 and C3 activation fragments | |
Sequence variations and protein expression levels of the two immune evasion proteins Gpm1 and Pra1 influence virulence of clinical Candida albicans isolates | |
Serum FHR1 binding to necrotic-type cells activates monocytic inflammasome and marks necrotic sites in vasculopathies | |
Staphylococcus aureus proteins Sbi and Efb recruit human plasmin to degrade complement C3 and C3b | |
Streptococcus pneumoniae von Patienten mit hämolytisch-urämischem Syndrom schädigen Endothelzellen durch aktiviertes Plasmin und inhibieren das Komplementsystem | |
Successful discontinuation of eculizumab under immunosuppressive therapy in DEAP-HUS. | |
A synthetic protein as efficient multitarget regulator against complement over-activation | |
Tef1 ist ein multifaktorielles Protein von Candida albicans und moduliert die angeborene und adaptive Immunreaktion | |
Thrombotic microangiopathies: new insights and new challenges | |
Translation Elongation Factor Tuf of Acinetobacter baumannii Is a Plasminogen-Binding Protein | |
Tuf of Streptococcus pneumoniae is a surface displayed human complement regulator binding protein | |
Untersuchung der Stabilität der zellulären DNA, des DNA-Turnovers, und Konzepts der spezifischen Aktivität bei verschiedenen eukaryontischen Zellsystemen | |
The vesicle transport protein Vac1p is required for virulence of Candida albicans | |
Where next with atypical hemolytic uremic syndrome? | |
Yersinia enterocolitica YadA mediates complement evasion by recruitment and inactivation of C3 products |