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Urs Christen Wikidata

Christen, Urs 1966- German National Library

VIAF ID: 120842817 (Personal)

Permalink: http://viaf.org/viaf/120842817

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Adenovirus E3 MHC inhibitory genes but not TNF/Fas apoptotic inhibitory genes expressed in beta cells prevent autoimmune diabetes. Wikidata
Apoptosis of autoreactive CD8 lymphocytes as a potential mechanism for the abrogation of type 1 diabetes by islet-specific TNF-alpha expression at a time when the autoimmune process is already ongoing. Wikidata
Blockade but not overexpression of the junctional adhesion molecule C influences virus-induced type 1 diabetes in mice. Wikidata
CD4+ T cells are required for secondary expansion and memory in CD8+ T lymphocytes. Wikidata
Cost of institute was small change to Roche Wikidata
Do viral infections protect from or enhance type 1 diabetes and how can we tell the difference? Wikidata
Halothane metabolism: the dihydrolipoamide acetyltransferase subunit of the pyruvate dehydrogenase complex molecularly mimics trifluoroacetyl-protein adducts Wikidata
Immune response to a recombinant human TNFR55-IgG1 fusion protein: auto-antibodies in rheumatoid arthritis (RA) and multiple sclerosis (MS) patients have neither neutralizing nor agonist activities Wikidata
Influence of the vitamin D plasma level and vitamin D-related genetic polymorphisms on the immune status of patients with type 1 diabetes: a pilot study. Wikidata
The kidney as a novel target tissue for protein adduct formation associated with metabolism of halothane and the candidate chlorofluorocarbon replacement 2,2-dichloro-1,1,1-trifluoroethane. Wikidata
Lipopolysaccharide is in close proximity to each of the proteins in its membrane receptor complex. transfer from CD14 to TLR4 and MD-2 Wikidata
Molecular mimicry in halothane hepatitis: biochemical and structural characterization of lipoylated autoantigens. Wikidata
Rat GTP cyclohydrolase I is a homodecameric protein complex containing high-affinity calcium-binding sites Wikidata
role of inflammatory factors in type 1 diabetes German National Library
Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Wikidata

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