C Tackenberg
Tackenberg, Christian 1978-
VIAF ID: 103402246 (Personal)
Permalink: http://viaf.org/viaf/103402246
Preferred Forms
- 100 0 _ ‡a C Tackenberg
- 100 1 _ ‡a Tackenberg, Christian ‡d 1978-
- 100 1 _ ‡a Tackenberg, Christian ‡d 1978-
4xx's: Alternate Name Forms (2)
5xx's: Related Names (1)
Works
Title | Sources |
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Active vaccination with ankyrin G reduces β-amyloid pathology in APP transgenic mice. | |
Alzheimer's in a dish - induced pluripotent stem cell-based disease modeling | |
APOE2, E3, and E4 differentially modulate cellular homeostasis, cholesterol metabolism, and inflammatory response in isogenic iPSC-derived astrocytes | |
Aβ-mediated spine changes in the hippocampus are microtubule-dependent and can be reversed by a subnanomolar concentration of the microtubule-stabilizing agent epothilone D | |
Calcium flux-independent NMDA receptor activity is required for Aβ oligomer-induced synaptic loss | |
Characterization of the Blood Brain Barrier Disruption in the Photothrombotic Stroke Model | |
Divergent pathways mediate spine alterations and cell death induced by amyloid-beta, wild-type tau, and R406W tau. | |
Early accumulation of intracellular fibrillar oligomers and late congophilic amyloid angiopathy in mice expressing the Osaka intra-Aβ APP mutation | |
Effect of amyloid precursor protein and tau on dendritic spines and cell survival in an ex vivo model of Alzheimers's disease | |
Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system | |
Genetic ablation of the p66Shc adaptor protein reverses cognitive deficits and improves mitochondrial function in an APP transgenic mouse model of Alzheimer's disease. | |
NMDA receptor subunit composition determines beta-amyloid-induced neurodegeneration and synaptic loss. | |
Oxidative stress and altered mitochondrial protein expression in the absence of amyloid-β and tau pathology in iPSC-derived neurons from sporadic Alzheimer's disease patients. | |
A Practical Guide to the Automated Analysis of Vascular Growth, Maturation and Injury in the Brain | |
The secreted APP ectodomain sAPPα, but not sAPPβ, protects neurons against Aβ oligomer-induced dendritic spine loss and increased tau phosphorylation | |
Stem Cell Therapy for Repair of the Injured Brain: Five Principles | |
Tau aggregation and progressive neuronal degeneration in the absence of changes in spine density and morphology after targeted expression of Alzheimer's disease-relevant tau constructs in organotypic hippocampal slices. | |
Xeno-free induced pluripotent stem cell-derived neural progenitor cells for in vivo applications |