Stephen Dunnett British neuroscientist
Dunnett, Stephen B.
Dunnett, S. B. (Stephen B.)
Dunnett, S. B.
Dunnett, Stephen B. 1950-
Dunnett, S. B. (Stephen B.), 1950-
VIAF ID: 19846768 (Personal)
Permalink: http://viaf.org/viaf/19846768
Preferred Forms
- 200 _ | ‡a Dunnett ‡b Stephen B.
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- 100 1 _ ‡a Dunnett, S. B.
- 100 1 _ ‡a Dunnett, S. B.
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- 100 1 _ ‡a Dunnett, S. B. ‡q (Stephen B.)
- 100 1 _ ‡a Dunnett, S. B. ‡q (Stephen B.)
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- 100 1 _ ‡a Dunnett, Stephen B.
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- 100 1 _ ‡a Dunnett, Stephen B. ‡d 1950-
- 100 0 _ ‡a Stephen Dunnett ‡c British neuroscientist
4xx's: Alternate Name Forms (11)
Works
Title | Sources |
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Animal models of movement disorders | |
Brain damage : brain repair | |
Brain research bulletin. | |
Dopamine, 2005: | |
Évaluation du potentiel thérapeutique des stratégies de remplacement cellulairedans un modèle de lésion corticale chez la souris : transplantation neuronale etmobilisation des cellules souches endogènes | |
Functional neural transplantation | |
Huntington’s Disease | |
Porcine neural xenografts in the immunocompetent rat: immune response following grafting of expanded neural precursor cells. | |
Pre-treatment with dopamine agonists influence L-dopa mediated rotations without affecting abnormal involuntary movements in the 6-OHDA lesioned rat. | |
Preface | |
Primary and stem cell therapies for brain repair | |
Proactive interference effects on short-term memory in rats: I. Basic parameters and drug effects. | |
Prospects for new restorative and neuroprotective treatments in Parkinson's disease | |
Re-examining the ontogeny of substantia nigra dopamine neurons. | |
Regulatory impairments following selective 6-OHDA lesions of the neostriatum | |
Reprogramming the diseased brain. | |
Response disinhibition on a delayed matching to position task induced by amphetamine, nicotine and age | |
Reverse transcription of inserted DNA in a monkey gives us ANDi. | |
Rewiring the Parkinsonian brain | |
Role of corticostriatal and nigrostriatal inputs in malonate-induced striatal toxicity. | |
The search for genetic mouse models of prodromal Parkinson's disease. | |
Septal transplants restore maze learning in rats with fornix-fimbria lesions. | |
Serotonin hyperinnervation after foetal nigra or raphe transplantation in the neostriatum of adult rats | |
Spatial learning and motor deficits in aged rats. | |
Spatially and temporally restricted chemoattractive and chemorepulsive cues direct the formation of the nigro-striatal circuit | |
Spontaneous and drug-induced rotation following localized 6-hydroxydopamine and kainic acid-induced lesions of the neostriatum. | |
The "staircase test": a measure of independent forelimb reaching and grasping abilities in rats | |
Striatal graft projections are influenced by donor cell type and not the immunogenic background. | |
Striatal grafts alleviate bilateral striatal lesion deficits in operant delayed alternation in the rat. | |
Striatal grafts in rats with unilateral neostriatal lesions--I. Ultrastructural evidence of afferent synaptic inputs from the host nigrostriatal pathway. | |
Striatal grafts in rats with unilateral neostriatal lesions--III. Recovery from dopamine-dependent motor asymmetry and deficits in skilled paw reaching. | |
Striatal lesions in the mouse disrupt acquisition and retention, but not implicit learning, in the SILT procedural motor learning task. | |
Striatal neurons in striatal grafts are derived from both post-mitotic cells and dividing progenitors | |
Subthalamic nucleus lesions induce deficits as well as benefits in the hemiparkinsonian rat. | |
Subtle but progressive cognitive deficits in the female tgHD hemizygote rat as demonstrated by operant SILT performance. | |
Surveying the literature from animal experiments. | |
Survival and differentiation of rat and human epidermal growth factor-responsive precursor cells following grafting into the lesioned adult central nervous system. | |
The survival of neural precursor cell grafts is influenced by in vitro expansion. | |
Survival of nigral grafts within the striatum of marmosets with 6-OHDA lesions depends critically on donor embryo age. | |
Synaptic input and local output of dopaminergic neurons in grafts that functionally reinnervate the host neostriatum. | |
Synaptic relationships between cortical and dopaminergic inputs and intrinsic GABAergic systems within intrastriatal striatal grafts. | |
The T cell oncogene Tal2 is necessary for normal development of the mouse brain | |
Targeting delivery in Parkinson's disease | |
Therapeutical potential of mouse embryonic stem cells-derived dopaminergic neurons in a mouse model of Parkinson's disease. | |
Tissue transplants for PD. | |
Translation of cell therapies to the clinic: characteristics of cell suspensions in large-diameter injection cannulae. | |
Translation to clinical application | |
Transplantation of expanded neural precursor cells from the developing pig ventral mesencephalon in a rat model of Parkinson's disease | |
Transplantation site influences the phenotypic differentiation of dopamine neurons in ventral mesencephalic grafts in Parkinsonian rats | |
Trophic mechanisms are not enough | |
Unilateral 6-OHDA lesions induce lateralised deficits in a 'skinner box' operant choice reaction time task in rats. | |
Unilateral lesions of the dorsal striatum in rats disrupt responding in egocentric space. | |
Unilateral lesions of the medial agranular cortex impair responding on a lateralised reaction time task. | |
The use of rodent skilled reaching as a translational model for investigating brain damage and disease. | |
Validation of New Models and Treatments of Parkinson’s Disease in the Non-Human Primate. | |
Validation of the l-dopa-induced dyskinesia in the 6-OHDA model and evaluation of the effects of selective dopamine receptor agonists and antagonists | |
What helps can also hinder: A dissociation in the acute effect of levodopa treatment on motor and cognitive functions | |
মুখবন্ধ |