Hodgson, S.V.
Shirley Hodgson British geneticist
Hodgson, Shirley V.
Hodgson, S. V., 1945-
Hodgson, Shirley Victoria
VIAF ID: 82923 (Personal)
Permalink: http://viaf.org/viaf/82923
Preferred Forms
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- 100 1 _ ‡a Hodgson, S. V.
- 100 1 _ ‡a Hodgson, S. V.
- 100 1 _ ‡a Hodgson, S. V.
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- 100 1 _ ‡a Hodgson, Shirley V.
- 100 1 _ ‡a Hodgson, Shirley V.
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- 100 1 _ ‡a Hodgson, Shirley Victoria
- 100 0 _ ‡a Shirley Hodgson ‡c British geneticist
4xx's: Alternate Name Forms (23)
Works
Title | Sources |
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Allelic loss at SMAD4 in polyps from juvenile polyposis patients and use of fluorescence in situ hybridization to demonstrate clonal origin of the epithelium | |
Association between polymorphisms of the GPX1 gene and second primary tumours after index squamous cell cancer of the head and neck | |
Breast cancer risk and 6q22.33: combined results from Breast Cancer Association Consortium and Consortium of Investigators on Modifiers of BRCA1/2 | |
Cancer genetics services in Europe | |
Changes in psychological distress after cancer genetic counselling: a comparison of affected and unaffected women | |
A complex endocrine conundrum | |
Comprehensive genetic assessment of the ESR1 locus identifies a risk region for endometrial cancer | |
Diagnosis and Management of Hereditary Thyroid Cancer | |
DNA glycosylases involved in base excision repair may be associated with cancer risk in BRCA1 and BRCA2 mutation carriers | |
Ethical, social and economic issues in familial breast cancer: a compilation of views from the E.C. Biomed II Demonstration Project | |
Exploring the link between MORF4L1 and risk of breast cancer | |
Familial breast and ovarian cancer : genetics, screening, and management | |
Fine-mapping of the HNF1B multicancer locus identifies candidate variants that mediate endometrial cancer risk | |
Genetic Risk Score Mendelian Randomization Shows that Obesity Measured as Body Mass Index, but not Waist:Hip Ratio, Is Causal for Endometrial Cancer | |
Genetic testing and common disorders in a public health framework: how to assess relevance and possibilities. Background Document to the ESHG recommendations on genetic testing and common disorders | |
Genome-wide allelotyping of 104 Finnish colorectal cancers reveals an excess of allelic imbalance in chromosome 20q in familial cases | |
Germline BRCA mutation and outcome in young-onset breast cancer (POSH): a prospective cohort study | |
Germline BRCA mutations are associated with higher risk of nodal involvement, distant metastasis, and poor survival outcomes in prostate cancer | |
Germline CDKN1B/p27Kip1 mutation in multiple endocrine neoplasia | |
Germline mutations but not somatic changes at the MYH locus contribute to the pathogenesis of unselected colorectal cancers. | |
Guidelines for follow-up of women at high risk for inherited breast cancer: consensus statement from the Biomed 2 Demonstration Programme on Inherited Breast Cancer | |
Guidelines for the clinical management of familial adenomatous polyposis (FAP). | |
Haplotype structure in Ashkenazi Jewish BRCA1 and BRCA2 mutation carriers | |
Identification of 12 new susceptibility loci for different histotypes of epithelial ovarian cancer | |
Identification of a C/G polymorphism in the promoter region of the BRCA1 gene and its use as a marker for rapid detection of promoter deletions | |
Identification of ten variants associated with risk of estrogen-receptor-negative breast cancer | |
Inherited susceptibility to cancer, 1998: | |
Insurance implications for individuals with a high risk of breast and ovarian cancer in Europe | |
Large genomic deletions in AIP in pituitary adenoma predisposition. | |
Late toxicity is not increased in BRCA1/BRCA2 mutation carriers undergoing breast radiotherapy in the United Kingdom. | |
Low prevalence of germline BRCA1 mutations in early onset breast cancer without a family history | |
Lymphocyte telomere length is long in BRCA1 and BRCA2 mutation carriers regardless of cancer-affected status | |
Mendelian randomization analyses suggest a role for cholesterol in the development of endometrial cancer | |
Meta-analysis of genome-wide association studies identifies common susceptibility polymorphisms for colorectal and endometrial cancer near SH2B3 and TSHZ1. | |
Mutation cluster region, association between germline and somatic mutations and genotype-phenotype correlation in upper gastrointestinal familial adenomatous polyposis | |
Mutation-Specific Survival of Inherited Breast Cancer | |
Mutations and alternative splicing of theBRCA1 gene in UK breast/ovarian cancer families | |
Mutations in GATA2 cause primary lymphedema associated with a predisposition to acute myeloid leukemia (Emberger syndrome) | |
No evidence of RET germline mutations in familial pituitary adenoma. | |
No evidence that GATA3 rs570613 SNP modifies breast cancer risk | |
An original phylogenetic approach identified mitochondrial haplogroup T1a1 as inversely associated with breast cancer risk in BRCA2 mutation carriers | |
Peutz-Jeghers disease: most, but not all, families are compatible with linkage to 19p13.3. | |
A practical guide to human cancer genetics | |
Risk estimation as a decision-making tool for genetic analysis of the breast cancer susceptibility genes. EC Demonstration Project on Familial Breast Cancer | |
Risks of Breast, Ovarian, and Contralateral Breast Cancer for BRCA1 and BRCA2 Mutation Carriers | |
The rs10993994 risk allele for prostate cancer results in clinically relevant changes in microseminoprotein-beta expression in tissue and urine | |
Screening SMAD1, SMAD2, SMAD3, and SMAD5 for germline mutations in juvenile polyposis syndrome | |
Sequence changes in predicted promoter elements of STK11/LKB1 are unlikely to contribute to Peutz-Jeghers syndrome | |
Targeted prostate cancer screening in men with mutations in BRCA1 and BRCA2 detects aggressive prostate cancer: preliminary analysis of the results of the IMPACT study | |
UBE2QL1 is disrupted by a constitutional translocation associated with renal tumor predisposition and is a novel candidate renal tumor suppressor gene | |
Utilisation of prophylactic mastectomy in 10 European centres | |
What motivates interest in attending a familial cancer genetics clinic? | |
Whole-gene APC deletions cause classical familial adenomatous polyposis, but not attenuated polyposis or "multiple" colorectal adenomas |