Ole Holger Petersen
Petersen, O. H. (Ole Holger)
Petersen, Ole H.
Petersen, Ole Holger
Petersen, Ole Holger, 1943-
Petersen, O.H.
Petersen, Ole H. 1943-
VIAF ID: 33120267 (Personal)
Permalink: http://viaf.org/viaf/33120267
Preferred Forms
-
100 0 _ ‡a Ole Holger Petersen
-
200 _ | ‡a Petersen ‡b Ole Holger
-
-
-
100 1 _ ‡a Petersen, O. H. ‡q (Ole Holger)
-
-
100 1 _ ‡a Petersen, O. H. ‡q (Ole Holger)
-
-
100 1 _ ‡a Petersen, Ole H.
-
-
100 1 _ ‡a Petersen, Ole H. ‡d 1943-
-
-
100 1 _ ‡a Petersen, Ole Holger
-
-
4xx's: Alternate Name Forms (18)
5xx's: Related Names (5)
- 510 2 _
‡a
Copenhagen
- 510 2 _
‡a
School of Biosciences
- 510 2 _
‡a
University of Liverpool
- 510 2 _
‡a
University of Liverpool
‡4
affi
‡4
https://d-nb.info/standards/elementset/gnd#affiliation
‡e
Affiliation
- 510 2 _
‡a
University of Wales
‡g
Cardiff
‡b
School of Biosciences
‡4
affi
‡4
https://d-nb.info/standards/elementset/gnd#affiliation
‡e
Affiliation
Works
Title | Sources |
---|---|
25 years of giga-seal patch clamping |
![]() ![]() |
ABT-199 (Venetoclax), a BH3-mimetic Bcl-2 inhibitor, does not cause Ca2+ -signalling dysregulation or toxicity in pancreatic acinar cells |
![]() |
Acetylcholine-induced ion transports involved in the formation of saliva |
![]() ![]() ![]() ![]() |
The ARRIVE guidelines 2.0: updated guidelines for reporting animal research |
![]() |
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis. |
![]() |
Both RyRs and TPCs are required for NAADP-induced intracellular Ca²⁺ release |
![]() |
Caffeine inhibits the agonist-evoked cytosolic Ca2+ signal in mouse pancreatic acinar cells by blocking inositol trisphosphate production |
![]() |
Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2 |
![]() |
Calcium and ATP control multiple vital functions |
![]() |
Calcium Measurement Methods |
![]() ![]() |
Control of enzyme secretion by non-cholinergic, non-adrenergic nerves in guinea pig pancreas |
![]() |
The Control of secretion, 1982, c1981: |
![]() ![]() ![]() ![]() |
The dependence of caerulein-evoked pancreatic fluid secretion on the extracellular calcium concentration |
![]() |
The dependence of the transmembrane salivary secretory potential on the external potassium and sodium concentration |
![]() |
EarLy Elimination of Fatty Acids iN hypertriglyceridemia-induced acuTe pancreatitis (ELEFANT trial): Protocol of an open-label, multicenter, adaptive randomized clinical trial |
![]() |
The effects of Ca2+ buffers on cytosolic Ca2+ signalling |
![]() |
The electrophysiology of gland cells |
![]() ![]() |
Endoscopic sphincterotoMy for delayIng choLecystectomy in mild acute biliarY pancreatitis (EMILY study): protocol of a multicentre randomised clinical trial |
![]() |
Extracellular but not intracellular application of peptide hormones activates pancreatic acinar cells |
![]() |
Galactose protects against cell damage in mouse models of acute pancreatitis |
![]() |
High versus low energy administration in the early phase of acute pancreatitis |
![]() |
The importance of extracellular sodium and potassium for acetylcholine-evoked salivary secretion |
![]() |
Inhibition of Salivary Secretion and Secretory Potentials by g-Strophantin, Dinitrophenol and Cyanide |
![]() |
Intracellular Ca2+ injection causes membrane hyperpolarization and conductance increase in lacrimal acinar cells |
![]() |
Ionic currents across pancreatic acinar cell membranes and their role in fluid secretion |
![]() |
Landmarks in intracellular signalling |
![]() ![]() |
Lecture notes, human physiology |
![]() ![]() ![]() |
Measuring calcium and calmodulin inside and outside cells, 2001: |
![]() ![]() ![]() ![]() ![]() ![]() ![]() |
The mechanism by which cholecystokinin peptides excite their target cells. Review |
![]() |
Mechanisms of action of hormonal and neuronal peptides on exocrine gland cells |
![]() |
Mechanisms of cellular calcium oscillations in secretory cells |
![]() |
Membrane potential and conductance changes evoked by hormones and neurotransmitters in mammalian exocrine gland cells |
![]() |
Mitochondrial injury in pancreatitis |
![]() |
NAADP mobilizes Ca2+ from a thapsigargin-sensitive store in the nuclear envelope by activating ryanodine receptors |
![]() |
Neural and hormonal control of membrane conductance in the pig pancreatic acinar cell |
![]() |
Nitric oxide signals are interlinked with calcium signals in normal pancreatic stellate cells upon oxidative stress and inflammation |
![]() |
Observational longitudinal multicentre investigation of acute pancreatitis (GOULASH PLUS): follow-up of the GOULASH study, protocol |
![]() |
One or Two Ca2+ Stores in the Neuronal Endoplasmic Reticulum? |
![]() |
Pancreatic acinar cells: effects of micro-ionophoretic polypeptide application on membrane potential and resistance |
![]() |
Pancreatic acinar cells: the effect of carbon dioxide, ammonium chloride and acetylcholine on intercellular communication |
![]() |
Pancreatitis and calcium signalling: report of an international workshop. |
![]() |
Parotid acinar cells: ionic dependence of acetylcholine-evoked membrane potential changes |
![]() |
Patch-clamp study of rubidium and potassium conductances in single cation channels from mammalian exocrine acini |
![]() |
Quantification of Ca2+-activated K+ channels under hormonal control in pig pancreas acinar cells |
![]() |
The relationship between stimulation-induced potassium release and amylase secretion in the mouse parotid |
![]() |
Reproducibility - again |
![]() |
SARS-CoV-2 S Protein Subunit 1 Elicits Ca<sup>2+</sup> Influx - Dependent Ca<sup>2+</sup> Signals in Pancreatic Stellate Cells and Macrophages <i>In Situ</i> |
![]() |
The Secretion of Sodium and Potassium in Cat Submandibular Saliva during the First Period after Start of Stimulation |
![]() |
Secretory mechanisms of exocrine glands : proceedings of the Alfred Benzon Symposium VII, held at the premises of the Royal Danish Academy of Sciences and Letters, Copenhagen, 9-12 September 1973 |
![]() ![]() ![]() |
Single calcium-dependent cation channels in mouse pancreatic acinar cells |
![]() |
Spatial and temporal distribution of agonist-evoked cytoplasmic Ca2+ signals in exocrine acinar cells analysed by digital image microscopy |
![]() |
Spotlight on the background actors |
![]() |
Stimulus-secretion coupling: cytoplasmic calcium signals and the control of ion channels in exocrine acinar cells |
![]() |
Stimulus-secretion coupling in mammalian salivary glands |
![]() |
Substance P and bombesin elevate cytosolic Ca2+ by different molecular mechanisms in a rat pancreatic acinar cell line |
![]() |
Thyrotropin controls cyclic nucleotide metabolism of thyroid follicular cells without affecting membrane potential or input resistance |
![]() |
Transport of calcium in the perfused submandibular gland of the cat |
![]() |
Voltage and Ca2+-activated K+ channel in baso-lateral acinar cell membranes of mammalian salivary glands |
![]() |