anticarcinogenic potential of dietary natural compounds on colorectal carcinoma effects on signalling pathways related to proliferation and cell death |
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Decoding E-cadherin glycans functions in cancer from functional glycomics to clinical applications |
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Diagnose cancer-causative E-cadherin missense mutations through molecular and morphological changes involved in cell extrusion |
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Diagnosticar mutações missense de caderina-E causadoras de cancro através de alterações moleculares e morfológicas envolvidas na extrusão celular |
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Evaluation of the use of helicobacter pylori genotypes and gastric microbiota markers to identify individuals at high risk of gastric carcinoma |
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Factors that contribute to the malignant transformation of e-cadherin mutant cells in HDGC study in drosophila, cell culture and tumours |
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Gastric carcinoma chromossomes and genes... |
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Identificação dos mecanismos moleculares que ligam as mutações da caderina-e à mobilidade celular |
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Identification of the molecular mechanisms linking e-cadherin mutations and cell motility |
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The importance of E-cadherin binding partners to evaluate the pathogenicity of E-cadherin missense mutations associated to HDGC. |
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Influence of ECM dynamic on gastric cancer stem-like properties |
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The interferon gamma receptor 1 |
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The interleukin-8-251*T/*A polymorphism is not associated with risk for gastric carcinoma development in a Portuguese population |
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The intracellular E-cadherin germline mutation V832 M lacks the ability to mediate cell-cell adhesion and to suppress invasion. |
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Karyotyping and DNA flow cytometry of an orchidoblastoma |
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KRAS mutation testing for predicting response to anti-EGFR therapy for colorectal carcinoma: proposal for an European quality assurance program. |
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KRASMutations and Anti–Epidermal Growth Factor Receptor Therapy in Colorectal Cancer With Lymph Node Metastases |
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Loss and recovery of Mgat3 and GnT-III Mediated E-cadherin N-glycosylation is a mechanism involved in epithelial-mesenchymal-epithelial transitions |
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Loss of functional E-cadherin renders cells more resistant to the apoptotic agent taxol in vitro |
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Luteolin, quercetin and ursolic acid are potent inhibitors of proliferation and inducers of apoptosis in both KRAS and BRAF mutated human colorectal cancer cells |
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Macrophages stimulate gastric and colorectal cancer invasion through EGFR Y(1086), c-Src, Erk1/2 and Akt phosphorylation and smallGTPase activity |
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O-mannosylation and N-glycosylation: two coordinated mechanisms regulating the tumour suppressor functions of E-cadherin in cancer. |
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Matrix metalloproteases as maestros for the dual role of LPS- and IL-10-stimulated macrophages in cancer cell behaviour |
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The mechanisms underlying MMR deficiency in immunodeficiency-related non-Hodgkin lymphomas are different from those in other sporadic microsatellite instable neoplasms |
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Microbial-based therapy of cancer: current progress and future prospects |
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Microsatellite instability in hyperplastic and adenomatous polyps of the stomach |
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Mixed lineage kinase 3 gene mutations in mismatch repair deficient gastrointestinal tumours |
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Model of the early development of diffuse gastric cancer in E-cadherin mutation carriers and its implications for patient screening |
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Molecular-based approaches to characterize the microbiome exploring the gastric cancer scenario |
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Molecular basis of the effect of radiotherapy on macrophage and on macrophage-cancer cell crosstalk |
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Molecular cloning of the synovial sarcoma-specific translocation (X;18)(p11.2;q11.2) breakpoint |
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Molecular targets and biological modifiers in gastric cancer |
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Mononucleotide precedes dinucleotide repeat instability during colorectal tumour development in Lynch syndrome patients |
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MUC1 gene polymorphism and gastric cancer--an epidemiological study. |
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mutator phenotype in sporadic gastric carcinomas causes and consequences |
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New target genes in endometrial tumors show a role for the estrogen-receptor pathway in microsatellite-unstable cancers. |
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The NMD mRNA surveillance pathway downregulates aberrant E-cadherin transcripts in gastric cancer cells and in CDH1 mutation carriers |
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NOD2/CARD15 and TNFA, But Not ILLB and ILLRN, are Associated With Crohnʼs Disease |
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Non-CDH1-Associated Familial Gastric Cancer and Epigenetics Factors |
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Novel expressed sequences obtained by means of a suppression subtractive hybridisation analysis from the 6q21 region that is frequently deleted in gastric cancer |
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P-cadherin functional role is dependent on E-cadherin cellular context: a proof of concept using the breast cancer model |
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P-cadherin role in normal breast development and cancer |
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p53 alterations in gastric carcinoma: |
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papel da regulação do transporte da Caderina-E na progressão do cancro epitelial |
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papel das mutações activantes dos genes KRAS e BRAF em carcinomas colrectais com e sem instabilidade de microssatélites |
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PCR amplification of DNA obtained from archived hematoxylin-eosin--and giemsa-stained breast cancer aspirates |
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Pesquisa de novos biomarcadores em cancro do estômago |
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The prevalence of PIK3CA mutations in gastric and colon cancer. |
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Preventing E-cadherin aberrant N-glycosylation at Asn-554 improves its critical function in gastric cancer |
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Proliferation and survival molecules implicated in the inhibition of BRAF pathway in thyroid cancer cells harbouring different genetic mutations. |
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Quantification of epigenetic and genetic 2nd hits in CDH1 during hereditary diffuse gastric cancer syndrome progression. |
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Quantification of mutant E-cadherin using bioimaging analysis of in situ fluorescence microscopy. A new approach to CDH1 missense variants. |
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Quantification of topological features in cell meshes to explore E-cadherin dysfunction |
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Rare Variants in the Epithelial Cadherin Gene Underlying the Genetic Etiology of Nonsyndromic Cleft Lip with or without Cleft Palate |
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Re. ‘Cellular phenotypes of differentiated-type adenocarcinomas and precancerous lesions of the stomach are dependent on the genetic pathways’ |
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Relevância clínica dos polimorfismos dos genes NOD2-CARD15, TNF-alfa, IL-beta e IL-1RN na doença de Crohn |
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role of BARAF and KRAS activating mutations in deficient and proficient MMR colorectal carcinomas |
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role of e-cadherin trafficking regulation in apithelial cancer progression |
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The role of N-acetylglucosaminyltransferase III and V in the post-transcriptional modifications of E-cadherin |
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Role of pathology in the identification of hereditary diffuse gastric cancer: report of a Portuguese family |
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Role of site-specific promoter hypomethylation in aberrant MUC2 mucin expression in mucinous gastric carcinomas |
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S100P is a molecular determinant of E-cadherin function in gastric cancer |
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Screening E-cadherin in gastric cancer families reveals germline mutations only in hereditary diffuse gastric cancer kindred |
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Searching for novel diagnostic-prognostic biomarkers in gastric cancer |
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Sequence diversity at the proximal 14q32.1 SERPIN subcluster: evidence for natural selection favoring the pseudogenization of SERPINA2. |
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Specific clinical and biological features characterize inflammatory bowel disease associated colorectal cancers showing microsatellite instability. |
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Sporadic gastric carcinomas with microsatellite instability display a particular clinicopathologic profile. |
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SRC inhibition prevents P-cadherin mediated signaling and function in basal-like breast cancer cells |
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Substantial reduction of the gastric carcinoma critical region at 6q16.3-q23.1 |
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Target gene mutational pattern in Lynch syndrome colorectal carcinomas according to tumour location and germline mutation |
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Tetra- and pentanucleotide short tandem repeat instability in gastric cancer. |
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Therapeutic targets associated to E-cadherin dysfunction in gastric cancer. |
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TP53 germline mutations in Portugal and genetic modifiers of age at cancer onset. |
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Transcription initiation arising from E-cadherin/CDH1 intron2: a novel protein isoform that increases gastric cancer cell invasion and angiogenesis |
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Tumor Necrosis Factor Alpha Extended Haplotypes and Risk of Gastric Carcinoma |
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Tumour selection advantage of non-dominant negative P53 mutations in homozygotic MDM2-SNP309 colorectal cancer cells |
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Unmasking the role of KRAS and BRAF pathways in MSI colorectal tumors. |
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WNT-inducible signaling pathway protein 3, WISP-3, is mutated in microsatellite unstable gastrointestinal carcinomas but not in endometrial carcinomas |
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