Lars Fugger
Fugger, Lars
VIAF ID: 43076184 (Personal)
Permalink: http://viaf.org/viaf/43076184
Preferred Forms
- 100 1 _ ‡a Fugger, Lars (sparse)
- 100 1 _ ‡a Fugger, Lars
- 100 1 _ ‡a Fugger, Lars
- 100 1 _ ‡a Fugger, Lars
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- 100 0 _ ‡a Lars Fugger
4xx's: Alternate Name Forms (2)
Works
Title | Sources |
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Antigen Targeting to Human HLA Class II Molecules Increases Efficacy of DNA Vaccination | |
APOBEC3G-induced hypermutation of human immunodeficiency virus type-1 is typically a discrete "all or nothing" phenomenon | |
Arne Svejgaard and immunogenetics in Denmark. | |
Association of MHC and rheumatoid arthritis. HLA-DR4 and rheumatoid arthritis: studies in mice and men | |
Autoimmunity's next top models. | |
Axonal injury in reverse | |
B cell tolerance and antibody production to the celiac disease autoantigen transglutaminase 2 | |
Bayesian analysis of genetic association across tree-structured routine healthcare data in the UK Biobank | |
Challenges, Progress, and Prospects of Developing Therapies to Treat Autoimmune Diseases | |
Characterization of T-cell receptor transgenic mice recognizing immunodominant HLA-DQ2.5-restricted gluten epitopes | |
Conflicting selective forces affect T cell receptor contacts in an immunodominant human immunodeficiency virus epitope | |
Constitutively active Lck kinase in T cells drives antigen receptor signal transduction | |
Control of autoimmunity by "epitope theft". | |
Convergent inputs from electrically and topographically distinct orexin cells to locus coeruleus and ventral tegmental area. | |
Crystal structure of HLA-DQ0602 that protects against type 1 diabetes and confers strong susceptibility to narcolepsy | |
Deletion of TASK1 and TASK3 channels disrupts intrinsic excitability but does not abolish glucose or pH responses of orexin/hypocretin neurons | |
Destructive processing by asparagine endopeptidase limits presentation of a dominant T cell epitope in MBP. | |
Direct and indirect control of orexin/hypocretin neurons by glycine receptors. | |
Dominant protection from HLA-linked autoimmunity by antigen-specific regulatory T cells | |
Efficient purification of unique antibodies using peptide affinity-matrix columns. | |
Elevated genetic risk for multiple sclerosis emerged in steppe pastoralist populations | |
Factors influencing success of clinical genome sequencing across a broad spectrum of disorders | |
From genes to function: the next challenge to understanding multiple sclerosis | |
Functional epistasis on a common MHC haplotype associated with multiple sclerosis | |
Genomewide Association Studies and Common Disease — Realizing Clinical Utility | |
HIV-1 adaptation to antigen processing results in population-level immune evasion and affects subtype diversification | |
HLA and disease - the molecular basis | |
The HLA-DRB1*15:01-restricted Goodpasture's T cell epitope induces GN. | |
The Human Cell Atlas White Paper | |
Humanized mouse models for organ-specific autoimmune diseases | |
Identifying cross-disease components of genetic risk across hospital data in the UK Biobank | |
Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice. | |
The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis. | |
In vivo enhancement of peptide display by MHC class II molecules with small molecule catalysts of peptide exchange. | |
Inflammation-related plasma and CSF biomarkers for multiple sclerosis | |
Inherited deficiency of mannan-binding lectin-associated serine protease 2. | |
Inhibitory Interplay between Orexin Neurons and Eating | |
Interleukin-17 production in central nervous system-infiltrating T cells and glial cells is associated with active disease in multiple sclerosis | |
Joint-specific and systemic autoreactivity in the development of inflammatory arthritis | |
Limited protective effect of the CCR5Delta32/CCR5Delta32 genotype on human immunodeficiency virus infection incidence in a cohort of patients with hemophilia and selection for genotypic X4 virus | |
Longitudinal and cross-sectional studies of HIV-1 RNA and DNA loads in blood and the female genital tract | |
Metabolism-independent sugar sensing in central orexin neurons. | |
MHC class II proteins and disease: a structural perspective | |
MHC II molecules in inflammatory diseases: interplay of qualities and quantities | |
Misfiring in multiple sclerosis: cerebellar channelopathy, a potential novel target? | |
Multiple sclerosis: insights into pathophysiology and novel therapeutic avenues | |
The nature of molecular recognition by T cells | |
Neuroinflammation - using big data to inform clinical practice | |
Novel synthetic amino acid copolymers that inhibit autoantigen-specific T cell responses and suppress experimental autoimmune encephalomyelitis | |
Opposing effects of HLA class I molecules in tuning autoreactive CD8+ T cells in multiple sclerosis | |
Orexin neurons as conditional glucosensors: paradoxical regulation of sugar sensing by intracellular fuels. | |
Pathogenic CD8 | |
A plasmid-encoded peptide from Staphylococcus aureus induces anti-myeloperoxidase nephritogenic autoimmunity | |
Please mind the gap: axonal transport deficits in multiple sclerosis neurodegeneration. | |
Recombinant TCR ligand induces tolerance to myelin oligodendrocyte glycoprotein 35-55 peptide and reverses clinical and histological signs of chronic experimental autoimmune encephalomyelitis in HLA-DR2 transgenic mice. | |
Recovery from COVID-19 in a B-cell-depleted multiple sclerosis patient | |
Redirecting therapeutic T cells against myelin-specific T lymphocytes using a humanized myelin basic protein-HLA-DR2-zeta chimeric receptor | |
Remarkably low affinity of CD4/peptide-major histocompatibility complex class II protein interactions | |
Resolving TYK2 locus genotype-to-phenotype differences in autoimmunity | |
The selection landscape and genetic legacy of ancient Eurasians | |
Severe B-cell-mediated CNS disease secondary to alemtuzumab therapy. | |
Spontaneous human squamous cell carcinomas are killed by a human cytotoxic T lymphocyte clone recognizing a wild-type p53-derived peptide | |
Stimulation of orexin/hypocretin neurones by thyrotropin-releasing hormone. | |
Structural and regulatory diversity shape HLA-C protein expression levels | |
Structure of HLA-A*0301 in complex with a peptide of proteolipid protein: insights into the role of HLA-A alleles in susceptibility to multiple sclerosis | |
Synthetic amino acid copolymers that bind to HLA-DR proteins and inhibit type II collagen-reactive T cell clones | |
Systematic classification of shared components of genetic risk for common human diseases | |
T cell-mediated autoimmune disease due to low-affinity crossreactivity to common microbial peptides | |
T cells and microglia as drivers of multiple sclerosis pathology | |
Tandem-pore K+ channels mediate inhibition of orexin neurons by glucose | |
Targeting ASIC1 in primary progressive multiple sclerosis: evidence of neuroprotection with amiloride | |
TCR transgenic mice that shed light on immune and environmental regulators in multiple sclerosis | |
Therapeutic vaccination of active arthritis with a glycosylated collagen type II peptide in complex with MHC class II molecules | |
Therapies for multiple sclerosis: translational achievements and outstanding needs | |
Tissue transglutaminase selectively modifies gliadin peptides that are recognized by gut-derived T cells in celiac disease. | |
TNF receptor 1 genetic risk mirrors outcome of anti-TNF therapy in multiple sclerosis | |
Tolerance to ingested deamidated gliadin in mice is maintained by splenic, type 1 regulatory T cells. | |
Tracking of proinflammatory collagen-specific T cells in early and late collagen-induced arthritis in humanized mice. | |
Use of calcium channel blockers and Parkinson's disease | |
The value of animal models for drug development in multiple sclerosis. | |
Visualization of myelin basic protein (MBP) T cell epitopes in multiple sclerosis lesions using a monoclonal antibody specific for the human histocompatibility leukocyte antigen (HLA)-DR2-MBP 85-99 complex | |
Weighing in on autoimmune disease: Big data tip the scale. |