Timo Hautala researcher (ORCID 0000-0001-7889-1614)
Hautala, Timo
VIAF ID: 279899718 (Personal)
Permalink: http://viaf.org/viaf/279899718
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100 1 _ ‡a Hautala, Timo (sparse)
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Hautala, Timo
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100 1 _ ‡a Hautala, Timo
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Timo Hautala
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researcher (ORCID 0000-0001-7889-1614)
4xx's: Alternate Name Forms (2)
Works
| Title | Sources |
|---|---|
| IL-2 may possess neuroprotective properties in glaucomatous optic neuropathy |
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| ADA2 deficiency: clonal lymphoproliferation in a subset of patients |
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| Adaptive or maladaptive response to adenoviral adrenomedullin gene transfer is context-dependent in the heart |
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| Adenoviral VEGF-A gene transfer induces angiogenesis and promotes bone formation in healing osseous tissues |
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| Adrenomedullin gene transfer induces neointimal apoptosis and inhibits neointimal hyperplasia in injured rat artery |
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| Aetiology of posterior uveitis in a tertiary centre in Finland |
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| Blood culture Gram stain and simple clinical categorisation can be used to identify patients at risk for delay in appropriate antibiotic treatment |
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| Clinical Candida krusei isolates remain susceptible during extensive exposure to antifungal drugs |
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| A cluster of Candida krusei infections in a haematological unit |
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| Combined immunodeficiency and hypoglycemia associated with mutations in hypoxia upregulated 1. |
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| Damaging heterozygous mutations in NFKB1 lead to diverse immunologic phenotypes |
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| Effect of first-line antifungal treatment on ocular complication risk in or yeast blood stream infection |
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| Elevated cerebrospinal fluid neopterin concentration is associated with disease severity in acute Puumala hantavirus infection |
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| Endostatin inhibits endochondral ossification |
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| Endostatin inhibits VEGF-A induced osteoclastic bone resorption in vitro |
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| Enrichment of rare variants in population isolates: single AICDA mutation responsible for hyper-IgM syndrome type 2 in Finland |
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| A Family With A20 Haploinsufficiency Presenting With Novel Clinical Manifestations and Challenges for Treatment |
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| Haploinsufficiency of A20 impairs protein-protein interactome and leads into caspase-8-dependent enhancement of NLRP3 inflammasome activation |
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| Heterozygous TLR3 Mutation in Patients with Hantavirus Encephalitis |
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| Human lysyl hydroxylase : cDNA cloning, chromosomal assignment, regulation of gene expression and analysis of mutations in patients with the type VIA Ehlers-Danlos syndrome |
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| Hypophyseal hemorrhage and panhypopituitarism during Puumala Virus Infection: Magnetic Resonance Imaging and detection of viral antigen in the hypophysis |
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| Inhibition of rubella virus replication by the broad-spectrum drug nitazoxanide in cell culture and in a patient with a primary immune deficiency |
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| Late-presenting vascular graft infection caused by Aspergillus in an immunocompetent patient |
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| Long-term hormonal follow-up after human Puumala hantavirus infection |
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| Major age group-specific differences in conjunctival bacteria and evolution of antimicrobial resistance revealed by laboratory data surveillance |
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| Minoxidil specifically decreases the expression of lysine hydroxylase in cultured human skin fibroblasts |
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| Nitazoxanide May Modify the Course of Progressive Multifocal Leukoencephalopathy. |
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| Plasma B-type natriuretic peptide (BNP) in acute Puumala hantavirus infection |
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| Prevalence, incidence and epidemiology of childhood uveitis |
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| Primary Immunodeficiency, a Possible Cause of Neutrophilic Necrotizing Dermatosis |
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| Prospective study on ocular findings in acute Puumala hantavirus infection in hospitalised patients |
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| Puumala virus RNA in cerebrospinal fluid in a patient with uncomplicated nephropathia epidemica |
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| Rubella persistence in epidermal keratinocytes and granuloma M2 macrophages in patients with primary immunodeficiencies |
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| Signs of general inflammation and kidney function are associated with the ocular features of acute Puumala hantavirus infection |
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| TLR3 deficiency in herpes simplex encephalitis: high allelic heterogeneity and recurrence risk |
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| Tonsillar granuloma associated with hypogammaglobulinemia |
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| True bacteremias caused by coagulase negative Staphylococcus are difficult to distinguish from blood culture contaminants |
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| Young male patients are at elevated risk of developing serious central nervous system complications during acute Puumala hantavirus infection |
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