Tegtmeyer, Nicole, 1976-....
Nicole Tegtmeyer
Tegtmeyer, Nicole
VIAF ID: 102718980 (Personal)
Permalink: http://viaf.org/viaf/102718980
Preferred Forms
- 100 0 _ ‡a Nicole Tegtmeyer
- 100 1 _ ‡a Tegtmeyer, Nicole
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- 100 1 _ ‡a Tegtmeyer, Nicole ‡d 1976-
- 100 1 _ ‡a Tegtmeyer, Nicole ‡d 1976-
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- 100 1 _ ‡a Tegtmeyer, Nicole, ‡d 1976-....
4xx's: Alternate Name Forms (3)
5xx's: Related Names (1)
- 551 _ _ ‡a Berlin
Works
Title | Sources |
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Bacterial type III effectors inhibit cell lifting by targeting integrin-linked kinase | |
Biochemical and functional characterization of Helicobacter pylori vesicles | |
c-Src and c-Abl kinases control hierarchic phosphorylation and function of the CagA effector protein in Western and East Asian Helicobacter pylori strains | |
Campylobacter jejuni serine protease HtrA plays an important role in heat tolerance, oxygen resistance, host cell adhesion, invasion, and transmigration | |
Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system | |
Delineation of a carcinogenic Helicobacter pylori proteome | |
Electron microscopic, genetic and protein expression analyses of Helicobacter acinonychis strains from a Bengal tiger | |
Extracellular secretion of protease HtrA from Campylobacter jejuni is highly efficient and independent of its protease activity and flagellum | |
H. pylori-Induced DNA Strand Breaks Are Introduced by Nucleotide Excision Repair Endonucleases and Promote NF-κB Target Gene Expression | |
Helicobacter pylori cell translocating kinase (CtkA/JHP0940) is pro-apoptotic in mouse macrophages and acts as auto-phosphorylating tyrosine kinase | |
Helicobacter pylori HtrA is a new secreted virulence factor that cleaves E-cadherin to disrupt intercellular adhesion | |
Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation. | |
Identification of E-cadherin signature motifs functioning as cleavage sites for Helicobacter pylori HtrA | |
Interplay of the Gastric Pathogen Helicobacter pylori with Toll-Like Receptors | |
Molecular pathogenesis and signal transduction by Helicobacter pylori | |
Molekulare Funktion von Cortactin in der Signaltransduktion und Pathogenese von gastrischen Epithelzellen | |
Overexpression of serine protease HtrA enhances disruption of adherens junctions, paracellular transmigration and type IV secretion of CagA by Helicobacter pylori | |
Peptide lineup against Gram-negative bacterial infection – first-in-class peptide inhibitor of H. pylori HtrA. | |
PKA-mediated phosphorylation of EPEC-Tir at serine residues 434 and 463: A novel pathway in regulating Rac1 GTPase function | |
The repA gene of the linear Yersinia enterocolitica prophage PY54 functions as a circular minimal replicon in Escherichia coli | |
An RGD helper sequence in CagL of Helicobacter pylori assists in interactions with integrins and injection of CagA | |
Role of Abl and Src family kinases in actin-cytoskeletal rearrangements induced by the Helicobacter pylori CagA protein | |
The role of serine protease HtrA in acute ulcerative enterocolitis and extra-intestinal immune responses during Campylobacter jejuni infection of gnotobiotic IL-10 deficient mice | |
Serine phosphorylation of cortactin controls focal adhesion kinase activity and cell scattering induced by Helicobacter pylori | |
The signaling pathway of Campylobacter jejuni-induced Cdc42 activation: Role of fibronectin, integrin beta1, tyrosine kinases and guanine exchange factor Vav2 | |
A small fibronectin-mimicking protein from bacteria induces cell spreading and focal adhesion formation | |
Subversion of host kinases: a key network in cellular signaling hijacked by Helicobacter pylori CagA. | |
Systematic analysis of phosphotyrosine antibodies recognizing single phosphorylated EPIYA-motifs in CagA of Western-type Helicobacter pylori strains | |
Transmigration route of Campylobacter jejuni across polarized intestinal epithelial cells: paracellular, transcellular or both? | |
Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression |